Medial displacement followed by quarter crack

Insulin triggers insulin-like growth factor receptors in the lamina. If rotation has not yet damaged the blood vessels significantly, high blood levels of insulin will cause rapid growth. When the dietary causes are addressed, the foot growth rate is normalized.
The first part is proven scientifically. The second part is from watching a lot of laminitis cases that have been properly addressed by diet.
Katy Watts

Long live the Grass Whisperer!

remember I asked if the horse was IR or had cushings? Katy Watts explained it.

Really Eric! Sorry you u get benched because of your narrow minded assertions.....

You have learned something.... foot mass

Get ya some this Eric! 1mm from coming through the foot. Medial displament will cause a medial toe flare. Arabs, count on the opposite. Your experiences are very narrow. Or your beliefs? Sorry Dude, you pulled the trigger....

Thanks Katy! But your assertions are not foundeed in mecahnics. They deal with metabolisms. Have seen Eruro's apply oil to feet everyday and jump meter 1.5, displacement. Horn integretiy,,,,,

Q: has anyone heard of the Jim Ferrie's Spiral Shoe?........apparently it addresses coffin joint issues....and he is in the Hall of Fame in Scotland

So.......

That's why I don't post much on farrier forums these days. Ya'll wanna talk about fixing stuff caused by metabolism with mechanics. Guess it keeps you busy doing what you like to do and what you get paid for.

Funny part is I asked you not Katy and you have no answer. The question I asked was about a foot with compromised blood supply, which she did point out " If rotation has not yet damaged the blood vessels significantly".

Well in my neck of the woods I'm lucky to have anything to trim off the ones I work on except a little heel in 8 weeks. Maybe these horses are not IR.

Do you have a link to the research?

No Dear your studies is/are appreciated by me and others!!!! But a determinated may or may not be metabolic. Ask Doc O,Grady or Scott Morrision the question I asked in San Antiotno(?), The room was quiet for 5 minunutes before a deflected answer,,,,,,,,,,,,,,mmmmmm

Or perhaps the foundered horses you work on have gone with their metabolisms ignored so long that the vasculature has been destroyed.

The journal papers are properly obscure and boring to read. Perhaps a summary prepared for laypeople would be more appropriate:
http://www.ahf-laminitis.org/2012/09/melody-delaat-laminitis-insulin.html

Other groups are working on this as well.
Equine Vet J. 2012 Jul 12.
Distribution of insulin receptor and insulin-like growth factor-1 receptor in the digitallaminae of mixed-breed ponies: An immunohistochemical study.

Burns TA, Watts MR, Weber PS, McCutcheon LJ, Geor RJ, Belknap JK.
Source

Department of Veterinary Clinical Sciences, The Ohio State University, Columbus, Ohio, USA Department of Large Animal Clinical Sciences, Michigan State University, East Lansing, Michigan, USA Department of Pathobiology and Diagnostic Investigation, Michigan State University, East Lansing, Michigan, USA.
Abstract

Reasons for performing study: Hyperinsulinaemia has been implicated in the pathogenesis of laminitis; however, laminar cell types responding to insulin remain poorly characterised. Objectives: To identify laminar cell types expressing insulin receptor(IRc) and/or insulin-like growth factor-1 receptor (IGF-1R); and to evaluate the effect of dietary nonstructural carbohydrate (NSC) on their expression. Methods: Mixed-breed ponies (n = 22) received a conditioning hay chop diet (NSC ∼6%); following acclimation, ponies were stratified into lean (n = 11, body condition score [BCS]≤4) or obese (n = 11, BCS ≥7) groups and each group further stratified to remain on the low NSC diet (n = 5 each for obese and lean) or receive a high NSC diet (total diet ∼42% NSC; n = 6 each for obese and lean) for 7 days. Laminar samples were collected at the end of the feeding protocol and stained immunohistochemically for IRc and IGF-1R. The number of IRc(+) cells was quantified;distribution of IGF-1R was qualitatively described. Laminar IRc content was assessed via immunoblotting. Results: The number of IRc(+) cells was greater in the laminae of high NSC ponies than low NSC ponies (P = 0.001); there was a positive correlation between the change in serum insulin concentration and number of IRc(+) cells (r(2) = 0.74; P<0.0001). No epithelial IRc(+) cells were observed; IRc(+) cells were absent from the deep dermis. Analysis of serial sections identified IRc(+) cells as endothelial cells. The distribution of IGF-1R was more extensive than that of IRc, with signal in vascular elements, epithelial cells and fibroblasts. Conclusions: Increased dietary NSC results in increased laminar endothelial IRc expression. Laminar keratinocytes do not express IRc, suggesting that insulin signalling in laminar epithelial cells must be mediated through other receptors (such as IGF-1R). Potential relevance: Manipulation of signalling downstream of IRc and IGF-1R may aid in treatment and prevention of laminitis associated with hyperinsulinaemia.

" This horse has had multiple laminitis episodes over the years. He is a 23 yr old gelding."

This screams of Cushing's aka PPID. You can continue to try and keep his feet from falling apart, or he can be tested and put on pergolide so the feet have a chance to gain some internal structural integrity.

YOU! and the good doctor Walsh have saved many a pony/horses on my books. See the Walsh studies of late fall gelding episodes people...

He's done presentations on it the last three summits. It is way over my pay grade but looks useful in a very very few cases. Looks like it would be very easy to do wrong. BTW the hall of fame is not in Scotland the presentations happen at summit every year.

David,
So basically the spiral shoe is like this side bone shoe, except that the taper comes back up a little on the lateral heel? Is the shoe fullered or just stamped, or what? Got a picture of one?